Srinivas RB, Kiran KRR, Naidu VG, Madhusudhana K, Agwane SB, Ramakrishna S, et al. Evaluation of antimicrobial, antioxidant and wound healing potentials of Holoptelea integrifolia. J Ethnopharmacol. 2008;115:249-256.
Khan HA, Ahmad A, Mehboob R. Nosocomial infections and their control strategies. Asian Pac J Trop Biomed 2015;5:509-514.
Calfee DP. Methicillin-resistant Staphylococcus aureus and vancomycin resistant enterococci, and other Gram-positives in healthcare. Curr Opin Infect Dis 2012; 25:385-394.
Gould IM, David MZ, Esposito S, Garau J, Lina G, Mazzei T, Peters G. New insights into meticillin-resistant Staphylococcus aureus (MRSA) pathogenesis, treatment and resistance. Int J Antimicrob Agents 2012; 39:96–104.
Klevens RM, Morrison MA, Nadle J, Petit S, Gershman K, Ray S, et al. Active bacterial core surveillance (ABCs) MRSA investigators. Invasive methicillin-resistant Staphylococcus aureus infections in the United States. JAMA 2007; 298:1763-1771.
Fischbach MA, Walsh CT. Antibiotics for emerging pathogens. Science 2007; 325:1089–1093.
Braine T. Race against time to develop new antibiotics. Bull World Health Organ 2011; 89:88–89.
Alanis AJ. Resistance to antibiotics: are we in the post-antibiotic era? Arch Med Res 2005; 36:697–705.
Marks LR, Clementi EA, Hakansson AP. The human milk protein-lipid complex HAMLET sensitizes bacterial pathogens to traditional antimicrobial agents. PLoS One 2012;7(8):e43514.
Marks LR, Clementi EA, Hakansson AP. Sensitization of Staphylococcus aureus to methicillin and other antibiotics in vitro and in vivo in the presence of HAMLET. PLoS One 2013;8:e63158
Svensson M, Hakansson A, Mossberg AK, Linse S, Svanborg C. Conversion of alpha-lactalbumin to a protein inducing apoptosis. Proc Natl Acad Sci USA 2000; 97: 4221–4226.
Kumar MS, Kirubanandan, S, Sripriya R, Sehgal PK. Triphala promotes healing of infected full-thickness dermal wound. J Surg Res 2008;144:94-101.
Qiu Z, Kwon AH, Kamiyama Y. Effects of plasma fibronectin on the healing of full-thickness skin wounds in streptozotocin-induced diabetic rats. J Surg Res 2007;138:64-70.
Peters EJ, Lipsky BA. Diagnosis and management of infection in the diabetic foot. Med Clin North Am. 2013; 97:911-946
Pattanayak SP, Sunita P. Wound healing, anti-microbial and antioxidant potential of Dendrophthoe falcata (L.f) Ettingsh. J Ethnopharmacol 2008; 120:241-247.
Dogan S, Demirer S, Kepenekci I, Erkek B, Kiziltay A, Hasirci N, et al. Epidermal growth factor containing wound closure enhances wound healing in non-diabetic and diabetic rats. Int Wound J 2009; 6:107–115.
Martin J, Zenilman M, Lazarus GS. Molecular microbiology: new dimensions for cutaneous biology and wound healing. J Invest Dermatol 2010; 130:38-48.
Berridge MJ. Cell Stress, Inﬂammatory Responses and Cell Death. Cell Signal Biol 2014 ;1:1-30.
Harper D, Young A, McNaught CE. The physiology of wound healing. Surgery (Oxford) 2014; 32:445-450.
Rodero M, Khosrotehrani K. Skin wound healing modulation by macrophages. Int J Clin Exp Pathol 2010; 3:643–653.
Elmore S. Apoptosis: a review of programmed cell death. Toxicol Pathol 2007; 35:495-516.
Wu YS, Chen SN. Apoptotic cell: linkage of inflammation and wound healing. Front Pharmacol 2014;5:1.
Bhan S, Mitra R, Arya AK, Pandey HP, Tripathi K. A Study on evaluation of apoptosis and expression of Bcl-2-Related marker in wound healing of streptozotocin-induced diabetic rats. ISRN Dermatol 7(2013), Article ID 739054, 6 pages.
Hockenbery D, Nunez G, Milliman C, Schreiber RD, Korsmeyer SJ. Bcl-2 is an inner itochondrialmembrane protein that blocks programmed cell death. Nature 1990; 348:334–336.
Jean SS, Hsueh PR. Current review of antimicrobial treatment of nosocomial pneumonia caused by multidrug-resistant pathogens. Expert Opin Pharmacother 2011;12: 2145–2148.
Rose WE, Poppens PT. Impact of biofilm on the in vitro activity of vancomycin alone and in combination with tigecycline and rifampicin against Staphylococcus aureus. J Antimicrob Chemother. 2009; 63:485–488.
Tang HJ, Chen CC, Ko WC, Yu WL, Chiang SR, Chuang YC. In vitro efficacy of antimicrobial agents against high-inoculum or biofilm-embedded methicillin resistant Staphylococcus aureus with vancomycin minimal inhibitory concentrations equal to 2 mug/ml (VA2-MRSA). Int J Antimicrob Agents 2011; 38:46–51.